Blue Waffle Disease – What is it?

It is a hard vaginal disease or a STD (Sexually Transmitted Disease) but unluckily there are not many information around it, especially recognized medical definitions. It has got it's name from the purple color it establishes to the vaginal surface area and the phrase “waffle” which is a jargon for the feminine reproductive organ.

The Symptoms of Blue Waffle

The symptoms can be replaceable to other vaginal infections but they are close to “vaginitis” or “trichomonas” . If an individual is infective the most general symptoms to feel would be itchiness in the vaginal area, puffiness, raised vaginal release, alterations in the coloration that change from blue to lilac. Still, the most severe thing here is to make the diagnosis by you. If you have any incertitudes that you are infective the greatest thing would be to go to see a doctor.

How Do You Get The Blue Waffle Infection?

As with many STDs the most frequent manner to induce this disease is to have vaginal sexual relation without using protection. In the present times, it is very essential to have not too many partners, and if you have then utilising a condom is a must. There are many other sexually carried infections in the world and new infections are being identified every day, so it is healthier to be safe than sorry.

Pictures of Blue Waffle

After searching the net looking for photos of blue waffle disease, you may have discovered that there is only one photo on all the blogs speaking just about it. It is a shocking image and it is not advised for minors to search for it since they might get disturbed. The lack of more images may direct us to a conclusion that this infection might be a scam. A pic with Photoshopped effects meant to develop a shock with the person who sees it.

Closing words

After a search there are many doubtfulnesses that give no solution. We don't have a medical explanation for the infection. Possibly the picture of the disease is intended to provoke the consciousness for STDs nowadays, when more young individuals are having their first intercourse at a very early age, when they are changing partners as much as they can.
Whatever it is, one thing is clear: If you think you have any sort of disease, go to talk with your doctor.

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images of systemic lupus erythematosus

10 thoughts on “Images Of Systemic Lupus Erythematosus

  1. Alanarama

    can anyone with lupus help please?
    i have recently been diagnose with raynauds syndrome which i understand can be a symtom of lupus.
    i have been to the doctors and bloods have been taken and sent off to find the cause of my raynauds i am just waiting for the results, but i am extremely worried. i have other symptoms of lupus too i just didnt realise they were symptoms of it.
    here goes
    ranaud’s symdrome
    white patches of skin near my eyes, genitalia, hips, and underarms
    butterfly rash
    aching knees (comes and goes)
    random nose bleeds (althouh i havent had one in a while)
    really red blistered toes
    chest pain when i breathe in sometimes(can be in my shoulder too)
    feelings of nausea or being sick for aparently no reason
    fever-i always feel freezing and have to wrap up but my boyfriend tells me iam really hot but i insist i am freezing cold
    abdominal pain and wierd feelings in my stomach
    red spots on my skin that come and go
    tingling and numbness in my arms mainly but sometimes in my legs
    i have been ill several times and they found exess protein in my unrine
    i get like blisters on my fingers and toes
    mostiof the time i want to do something but just feel “whats the point” or “im too tired”

    i think really what im wanting is your opinions if it could be lupus? or could it be something else? i guess i am just looking for reassurance. any help will be greatly appreciated, and any advice too, thanks xx

    1. Mazher

      Cellulitis is an acute infection of skin and soft tissues characterized by localized pain, swelling, tenderness, erythema, and warmth.

      Cellulitis usually follows a break in the skin, such as a fissure, cut, laceration, insect bite, or puncture wound. Facial cellulitis of odontogenic origin may also occur. Patients with toe web intertrigo and/or tinea pedis and those with lymphatic obstruction, venous insufficiency, pressure ulcers, and obesity are particularly vulnerable to recurrent episodes of cellulitis (Roujeau, 2004; Bjornsdottir, 2005; Roberts, 2005). Organisms on the skin and its appendages gain entrance to the dermis and multiply to cause cellulitis. The vast majority of cases are caused by Streptococcus pyogenes or Staphylococcus aureus. Occasionally, cellulitis may be caused by the emergence of subjacent osteomyelitis. Cellulitis may rarely result from the metastatic seeding of an organism from a distant focus of infection, especially in immunocompromised individuals. This is particularly common in cellulitis due to Streptococcus pneumoniae and marine vibrios.

      Cellulitis generally is a localized infection. Most patients treated appropriately recover completely. Mortality is rare (5%) but may occur in neglected cases or when cellulitis is due to highly virulent organisms (eg, Pseudomonas aeruginosa). Factors associated with an increased risk of death are the presence of concurrent illness (eg, congestive heart failure, morbid obesity, hypoalbuminemia, renal insufficiency) or complications (eg, shock) (Carratala, 2003).

      No predilection for either sex is usually reported, although a higher incidence among males has recently been reported in one study

      The clinical appearance of cellulitis is shown in Images 1-3.

      Involved sites are red, hot, swollen, and tender.
      Unlike erysipelas, the borders are not elevated or sharply demarcated.
      Lymphangitis, regional lymphadenopathy, or both may be present.
      Fever is common.
      In severe cases, patients may develop hypotension.
      Local suppuration may follow if therapy is delayed.
      Overlying skin may develop areas of necrosis.
      The most commonly involved site is the leg (Ellis Simonsen, 2006; Lazzarini, 2006).
      Perianal cellulitis due to group A streptococci is usually observed among children with perianal fissures. It is characterized by perianal erythema and pruritus, painful defecation, and bleeding in the stools (Spear, 1985).
      Pneumococcal facial cellulitis occurs primarily in young children who are at risk for pneumococcal bacteremia (Givner, 2000; Parada, 2000). It may manifest as two distinctive clinical syndromes.

      Extremity involvement in individuals with diabetes mellitus or substance abuse
      Head, neck, and upper torso involvement in individuals with systemic lupus erythematosus, nephrotic syndrome, or hematologic disorders


      In immunocompetent individuals, cellulitis is usually due to gram-positive aerobic cocci (eg, S aureus, S pyogenes) (Bisno, 1996; Guay, 2003; Carratala, 2003). Isolation of methicillin-resistant S aureus (MRSA) is steadily increasing (Eady, 2003). Bacterial strains may also show multiple resistance to other standard antibiotic treatments, including erythromycin.
      Recurrent staphylococcal cellulitis may occur in patients with nasal carriage of staphylococci and those with Job syndrome. S aureus is also the leading cause of soft tissue infections in persons who abuse injection drugs (Bassetti, 2004).
      Recurrent cellulitis due to streptococci may be observed in patients with chronic lymphedema (eg, from lymph node dissection, irradiation, Milroy disease, elephantiasis) (Bisno, 1996; Chmel, 1984). Streptococcal infections are also common in injection drug users (Sierra, 2006).
      Non–group A streptococci (ie, groups B, C, and G) are commonly implicated in cellulitis in patients with lymphatic obstruction or venectomy for coronary artery bypass graft (Baddour, 1982; Baddour, 1985).
      S pneumoniae is an uncommon cause of cellulitis in adults (Parada, 1999; Parada, 2000; Bachmeyer, 2006). Pneumococcal cellulitis may occur via bacteremia. In a review of pneumococcal skin infection in adults, all such patients had an underlying chronic illness or were immunocompromised by drug or alcohol abuse (Capdevila, 2003). Pneumococcal facial cellulitis occurs primarily in young children at risk for pneumococcal bacteremia (Patel, 1994; Givner, 2000).
      Patients who are immunocompromised with granulocytopenia, such as renal transplant recipients, may develop cellulitis due to infection with other organisms, including gram-negative bacilli (eg, Pseudomonas, Proteus, Serratia, Enterobacter, Citrobacter), anaerobes, other opportunistic pathogens (eg, Helicobacter cinaedi, Fusarium species), mycobacteria, and fungi (eg, Cryptococcus) (Anderson, 1992; Kiehlbauch, 1994; Horrevorts, 1994; Nucci, 2002; Guay, 2003; Yoo, 2003; Gupta, 2003; Seyahi, 2005). Preseptal cellulitis caused by dermatophytes is rarely observed, mostly in the pediatric age group (Rajalekshmi, 2003). Persistent cellulitis due to Cryptococcus neoformans infection has also been reported in a patient receiving renal dialysis (Suranyi, 2003).
      Escherichia coli may be responsible for cellulitis in patients with nephrotic syndrome (Asmar, 1987).
      Cellulitis from unusual bacterial species, including Enterococcus faecalis, Enterobacteriaceae, and Bacteroides and Clostridium species, may be observed following subcutaneous injections of illegal drugs (Dancer, 2002). If Clostridium species or other anaerobes cause the infection, crepitant cellulitis is often observed clinically.
      Other uncommon causes of cellulitis include Neisseria meningitidis; Pasteurella multocida, following animal bites; Aeromonas hydrophilia, following contact with fresh water; Streptococcus iniae, a fish pathogen causing infections in aquaculture farms; and Vibrio vulnificus, following contact with seawater. Cellulitis from marine vibrios in hepatopathic patients may also follow ingestion of contaminated raw oysters (Porras, 2001; Cartolano, 2003; Winner, 2003; Lau, 2003; Swartz, 2004, Falcon, 2005). Haemophilus influenzae has become a rare cause of buccal cellulitis in children after the introduction of the H influenzae type B vaccine (Branca, 2003; Lin, 2004

      Medical Care
      Patients with mild cases of cellulitis may be treated in an outpatient setting. Oral agents with activity against staphylococci and streptococci (eg, dicloxacillin or flucloxacillin, cephalexin, cefuroxime axetil, erythromycin, clindamycin, cotrimoxazole, amoxicillin/clavulanate) are usually effective for treatment of cellulitis in immunocompetent hosts (Roberts, 2005).

      Manage more severe disease initially with intravenous antibiotics in the hospital. This is also recommended in immunosuppressed individuals and in any patients with a clinically significant concurrent condition, including lymphedema and cardiac, hepatic, or renal failure.

      Elevating limbs with cellulitis expedites resolution of the swelling. Cool sterile saline dressings may be used to remove purulent discharge from any open lesion.

      Usually, cellulitis is presumed to be due to staphylococci or streptococci infection and is treated with antibiotics (eg, nafcillin, cefazolin). Other options in allergic patients include clindamycin or vancomycin. Ceftriaxone may be useful in the outpatient setting because it can be administered once daily (Seaton, 2005).
      Agents with a broader spectrum of activity are recommended in selected patients, such as diabetic patients (Swartz, 2004).
      More specific antibiotic therapy may be indicated in patients who develop cellulitis in special settings (eg, after a human or animal bite, exposure to potentially contaminated fresh water or seawater) (Swartz, 2004). Treatment of cellulitis caused by uncommon organisms, such as Vibrio species or gram-negative bacteria, should be individualized to those recovered organisms (Chuang, 1992). In general, these organisms require treatment with drugs other than those discussed above. For instance, cellulitis due to Vibrio infection may be treated with tetracyclines, chloramphenicol, or aminoglycosides.
      Cutaneous cellulitis and soft tissue infections due to community-acquired MRSA represent an emerging problem also among patients who lack traditional risk factors (Eady, 2003).

      In such cases, management with standard gram-positive antibiotics may be ineffective, also because concomitant multiresistance to other antibiotics widely used in common empiric therapy, including erythromycin, may occur. Bacterial strains are usually susceptible to clindamycin, gentamicin, rifampin, trimethoprim/sulfamethoxazole, and vancomycin.
      Moreover, a randomized, open-label, comparator-controlled, multicenter, multinational study has recently demonstrated the efficacy of linezolid therapy and its superiority to vancomycin in the management of skin and soft tissue infections, including cellulitis, due to MRSA (Weigelt, 2005). However, bacterial culture is still considered essential in order to determine the antibiotic susceptibility of the bacterial isolate and to adjust the systemic antimicrobial therapy according to sensitivity data.
      If mycologic investigations performed to rule out tinea pedis as a possible cause of recurrent episodes of cellulitis detect the presence of fungal infection in toe webs or feet, treatment with topical antifungals is recommended. With severe chronic changes or if onychomycosis is providing a source for repeated infection, oral antifungals such as itraconazole or terbinafine may be considered.

      Surgical Care
      Incision and drainage are indicated if suppuration has occurred.


      Consult an infectious diseases specialist for recommendations on appropriate antibiotic therapy.
      Consult a surgeon for drainage of any abscess and debridement of any devitalized tissue.

      Immobilization of the affected part may relieve pain.

      Read more here an

  2. Dishari

    My blood test of CRP is 33.24mg/l, What can I do ?
    I have SACRALIZATION of L5 on both side.ASO is 141.3 IU/ml,ANA- NEGATIVE,ANTI CCP 1.7,HLA-B27-NEGATIVE,MRI of LS spine- Mild diffuse annular bulge at L4-L5,compromising the bilateral I.V. neural foramina and compressing the bilateral exiting L4 nerve roots.I am male and 21 years.I have Pain. please help me. thank you.

    1. TweetyBird

      I would certainly hope that you wouldn’t have Viagra just lying around and you wouldn’t be able to get any without a prescription anyway. In any event, this is not the right drug to take.

      C-reactive protein is produced in the liver in response to tissue injury and inflammation and is found in many body fluids. Your levels of 33.42mg/L are high and i expect this is a quantitative value.

      Your ASO (antistreptolysin O) of 141.3 IU/mL measures the beta-hemolytic Streptococcus enzyme that this bacteria secret, which is streptolysin O. My lab uses a reference range of <100 IU/mL for a normal value. You may already know that the symbol "<" means "less than". You're a little outside the range of the normal upper limits. The ANA (antinuclear antibodies) should be negative as yours were. The ANA is a screening test for diagnosing systemic lupus erythematosus and other collagen diseases as well as scleroderma, rheumatoid arthritis, cirrhosis, leukemia, mono, lupus nephritis and malignancies. Perhaps no problem here but that remains to be seen. The anti-CCP (anti-cyclic citrullinated peptide antibody) is a sensitive and specific blood test that helps confirm the presence of rheumatoid arthritis before it's developed enough to show positives in other tests. So one could have both a positive anti-CCP with low levels and have a negative ANA. HLA-B27 (human leukocyte antigens) are on the surface membranes of leukocytes, platelets and tissue cells. The four main reasons for HLA testing are organ transplant, transfusion, paternity testing and disease association. Two of the diseases associated with HLA are rheumatoid arthritis and reactive arthritis Sacralization of L5 into S1 is rather common but often doesn't cause symptoms. Your MRI reflects an annular bulge at L4-L5. Annular means ring-shaped. The bulge is an intervertebral disc, one of the somewhat circular pads of fibrocartilage between each vertebrae of your spine that supports and cushions), it's ring-shaped (annular) and spread out (diffuse) and it's near the base of your lumbar spine (L5 is the base). The foramina (sing. foramen) are naturally occurring passageways through bones for nerves and blood vessels. Compromised means the herniated disc is partially blocking both foramina and compressing is obvious. The disc is impinging on the spinal nerves associated with L4 and 5, putting pressure on them and it's this....that's causing much of your pain. The sacralization may be causing pain of its own but I can't be sure of that. Overlapping pain can be hard to determine. But why, my friend, have you posted a question asking us for help with your pain???? Have you spoken to your orthopedic yet? Have you discussed your condition, labs and the implications of both? Because it seems to me that you have two things going on: (1) possible arthritis developing at the site of sacralization and (2) neuropathy (nerve pain) due to the herniation. The herniation is the priority. Your orthopedic may want to see if the disc repairs itself before recommending anything more drastic. For most people, it does improve. And if the herniation is going to get better, it will do so within 6 weeks. During that time, your doctor can help you by prescribing oral steroids like prednisone or methyprednisolone, recommending NSAIDs, ordering an epidural (a cortisone injection for the pain, can last for several months), physical therapy and there are other nonsurgical options. Your doctor may give the nonsurgical approach 6-12 weeks and if it doesn't work, a surgical approach is, at this point, not unreasonable. If your symptoms worsen before 6 weeks pass, surgical repair would be considered sooner. You just haven't mentioned having any doctor involved in this, yet some one had to order the labs and the imaging. This is the doctor you should be asking your questions. And this is the doctor with whom you should be discussing treatment options. Do this the right way, my friend. If you want the opinion of a health care provider, I AM one and I'm offering it to you. You'd be wise to take it to heart.

  3. nattydreddey

    The history of the classroom?
    I am generation X-er. I am curious to why schools (elementary through university) elimiated the good old fashioned chalk boards? No ignorant answers please!!

    1. Libby

      Modern whiteboards evolved from chalkboards.

      In the mid 1960s, the first whiteboards (also called markerboards) began to appear on the market. In classrooms, their widespread adoption didn’t occur until the late 1980s and early 1990s when concern over allergies and other potential health risks posed by chalk dust prompted the replacement of many blackboards with whiteboards. Beyond the human health aspects of chalk dust exposure, there are also potential electronic hazards. Devices such as computers and digital versatile disc (DVD) players stored inside classrooms can suffer damage from accumulated chalk dust. As the dust particles circulate throughout the room, cooling fans may draw them into the computers’ inner workings. As dust builds up on the motherboard and other heat-sensitive parts, the risk of overheating increases.
      * Whiteboard ink markings are less susceptible to external factors, such as water, because the ink adheres in a different manner than chalk does to chalkboards. Using markers does not generate the dust that comes from using and erasing chalk, allowing their use in areas containing dust-sensitive equipment. Some who are allergic to chalk or are asthmatic use whiteboards as an alternative.
      * A whiteboard can be used as the projecting medium for an overhead or video projector. This allows the person giving the presentation to fill in blanks, edit, underline and make comments by writing directly onto the whiteboard, which in turn shows through the projected image. Proper dry wipe boards are high gloss to enable the dry marker ink to be wiped off easily and high gloss surfaces will reflect the projector light, creating a so called “hot spot”, a glare back from the board. Semi-matt whiteboards are better suited for projection but more difficult to dry wipe clean.
      * A dry erase marker is easier to hold and write with. This can benefit persons with limited mobility in their hands, such as those affected by diseases such as arthritis or systemic lupus erythematosus. In addition, marking on a whiteboard takes less time, effort, and pressure than marking on a chalkboard.
      * Like chalkboards, whiteboards help to save paper.
      * When compared to a chalkboard a whiteboard can have significantly more colors because markers have a greater range of color than chalk.

  4. Jezz

    Hospital can’t diagnose whats wrong with me?
    What should i do? I’ve had a colonoscopy, gastroscopy, barium x-ray, ultrasound, blood tests, stool sample as well as various medications incase its IBS and they still can’t diagnose me and said theres little more they can do for me. Please answer im desperate. These are my symptoms. I’ve been ill for 3 months now with a stomach ache almost all the time, my appetite has gone, im struggling to eat and i’ve lost 30lb now. Im also exhausted all the time, im not normally a lazy person but im needing around 15 hours sleep a night now. Also sometimes i feel like i need the toilet but nothing or some mucus comes out (sorry i know thats sick) and i had diarrhoea until a few weeks ago, im not lactose intollerant, im not stressed and i’ve hardly been able to leave the house for 3 months. Im 21, dont smoke or drink and was very fit and healthy before i got this. What more could i do? Thanks a lot
    Thanks for all answers, its not mental health as i’ve never had any problems with that before and im not stressed,depressed or anything.
    And its not an ulcer and its too severe to be IBS.

    1. Maxi

      You really are desperate asking yahoo questions when the health service can’t find out what wrong.

      I would normally never answer this however, you have the similar symptoms that a friend had and it took them three years to diagnose her, then some doctors said it wasn’t ‘real’

      After lots of research, I believe it is a mental health issue ( no don’t get upset, if anyone has suffered any form of stress that is mental health and 99% have, mental health just scares us) No ‘real’ reason for the symptoms, but the pain, tiredness etc is very real.
      I found that NLP helped her a lot, regular meals, eat when hungry, not time, exercise and a Bioflow magnetic bracelet , go to bed when you are tired not by the time.
      I presume they tested for M.E. which was daub-ed the ‘juppy disease’

      “Fibroid myalgia” or fibromyalgia as it is better known is a common condition characterised by generalized pain and fatigue. This condition has nothing to do with fibroids.Fibromyalgia is considered a form of arthritis. The cause is unknown but theories implicating abnormal hypothalamic pituitary axis function or dysfunction of neurotransmitter pathways in the brain are currently popular.

      Approximately 2 per cent of the population has fibromyalgia. About 80 per cent of patients with fibromyalgia are women. While fibromyalgia may occur as a primary condition, it is also a secondary condition, occurring in as many as 30 per cent of patients with systemic lupus erythematosus and rheumatoid arthritis.

      Patients with fibromyalgia complain of generalised pain affecting both sides of the body and both the upper as well as lower part of the body.

      Pain tends to be aggravated by weather changes as well as by stress.

      While patients will complain of subjective joint swelling, objective swelling is absent. Here is a situation where magnetic resonance imaging which is able to detect minute amounts of synovitis that can help in the work up.

      Sleep disturbance occurs in almost all patients. Complaints of chronic fatigue and non restorative sleep (feeling as if they haven’t slept) are common. Sleep apnea may confound the situation.

      Tender trigger points are noted in all patients. A patient with 11 of 18 tender trigger points fulfills a major diagnostic criterion for the diagnosis of fibromyalgia. These trigger point tender areas are stereotypic meaning the same areas are tender in all patients with the diagnosis of FM.

      Other symptoms include migraine headache, decrease in short term memory, cognitive dysfunction, blurred or double vision, hypersensitivity to sound and smells, shortness of breath, chest pains, palpitations, irritable bowel, irritable bladder, painful menses, painful urination, multiple drug allergies, multiple sensitivities to chemicals.

      In fact, the types of symptoms a patient presents with may determine what type of physician that patient will seek out. For instance, patients with bowel symptoms may see a gastroenterologist. Patients with bladder irritability may see a urologist. Patients for whom depression is a problem may see a psychiatrist, and patient s who complain of migraine headaches may see a neurologist. Unexplained aches and pains will usually send a patient to see a rheumatologist or even an orthopedic surgeon.

      Laboratory testing will not be diagnostic. However, laboratory testing will help to exclude other conditions such as polymyalgia rheumatica, hypothyroidism, rheumatoid arthritis, systemic lupus erythematosus, etc., that might masquerade as fibromyalgia. FM is a diagnosis of exclusion so it is imperative that other possible causes of aches and pains are ruled out.

      Imaging tests may also be helpful in establishing the presence or absence of FM.

      Treatment must be individualised. Most patients will respond to a combination of non impact aerobic exercise (swimming, stationary bike, elliptical trainer), cognitive behavioral therapy, and medication.

      Medications that have been found to be helpful include tricyclic antidepressants in low doses, muscle relaxants such as cyclobenzaprine, also in low doses, and selective serotonin reuptake inhibitors (SSRIs)

      Other medicines such as gabapentin and tramadol may also be helpful. A more complete discussion of FM may be found elsewhere.

      More recently, drugs such as Mirapex have been used for their dopaminergic effect.

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